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srebpの例文

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  • Insulin induction of SREBP-1c is also involved in cholesterol metabolism.
  • Inhibition of SREBP by betulin decreased the biosynthesis of cholesterol and fatty acid.
  • SREBP-1c mediates MEF2C downregulation through a mechanism that remains to be determined.
  • Growth factors affect expression of NFE2L1 through a mTORC and SREBP-1 mediated pathway.
  • Its expression is induced by decreased sterol concentrations via sterol regulatory binding proteins ( SREBP ).
  • Nur77 is suggested to inhibit LXR and downstream SREBP-1c expression modulating hepatic lipid metabolism.
  • The nuclear localization of Lipin 1 is regulated by the SREBP )-dependent gene transcription.
  • In these vesicles, SCAP, dragging SREBP along with it, is transported to the Golgi.
  • Also, it binds SREBP by a series of consecutive WD repeats on its C-terminus.
  • FGF21 has been shown to repress the transcription of sterol regulatory element binding protein 1c ( SREBP-1c ).
  • A medium rich in branched-chain amino acids stimulates expression of the SREBP-1c gene via the S6K1 pathway.
  • Furthermore, SREBP-1c appears to be susceptible to diet, and thus it can be a target for nutritional intervention.
  • Unexpectedly, overexpression of SREBP-1c in HepG2 cells could also inhibit the endogenous FGF21 transcription by reducing FGF21 promoter activity.
  • LXRs regulate fatty acid synthesis by modulating the expression of sterol regulatory element binding protein-1c ( SREBP-1c ).
  • When cholesterol is present, SCAP undergoes a conformational change that prevents it from activating SREBP and cholesterol synthesis does not occur.
  • SREBP-1c regulates genes required for glucose metabolism and fatty acid and lipid production and its expression is regulated by insulin.
  • SREBP-1a regulates genes related to lipid and cholesterol production and its activity is regulated by sterol levels in the cell.
  • Their subsequent work shows how the SREBP pathway regulates expression of many genes that control lipid formation and metabolism and body fuel allocation.
  • SREBP-1c has also been shown to upregulate in a tissue specific manner the expression of PGC1alpha expression in brown adipose tissue.
  • By selectively inhibiting S1P, AEBSF can be used to characterize the downstream result of SREBP inhibition and its influence on cholesterol regulation.
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